Fig 1: OTR Gene expression level in the testes. OTR expression in the testes of the diabetic group of BALB/c mice was lower than in the sham and control groups. *P<0.05
Fig 2: a Oxtr mean DNA methylation (%) for all analyzed brain regions. b Methylation of specific CpG position (− 1279) in the PFC. c Protein expression (% of lesion animals) for all analyzed brain regions. Data is shown as mean ± SEM for lesion, sham-lesion, and control animals. Significant differences between sham-lesion and control animals are indicated by an asterisk (*, p < 0.05). Abbreviations: Ctx, sensorimotor cortex; NAc, nucleus accumbens; PFC, prefrontal cortex; S, striatum; T, thalamus
Fig 3: The view of PCR gel for OTR gene in the testes of control, diabetic, and sham groups of BALB/c mice
Fig 4: Effects of prenatal DEHP exposure and childhood exercise on the oxytocin pathway in postpartum dams. The results of ELISA showed that oxytocin levels in the plasma (a) and hypothalamus (b) were reduced by the effect of DEHP. (c) The result of qPCR showed a reduction of hypothalamic Oxtr mRNA influenced by the effect of DEHP exposure. (d) The result of the Western blot showed there was a significant effect of DEHP on reducing the hypothalamic OTR levels. Data are presented in mean ± SEM (n = 12 per group for plasma; n = 6 per group for hypothalamus). *: p < 0.05; ***: p < 0.005; ****: p < 0.001.
Fig 5: OTR immunoreactivity in the testis of the diabetic group. The mice were injected with STZ (100 mg/kg, IP). There was weak OTR immunoreactivity only in the interstitial area. Arrow: Interstitial area. Bar: 100 μm
Supplier Page from Abcam for Anti-Oxytocin Receptor antibody